Excess glucose is stored in large branched molecules of:
A. glycogen in animals.
B. starch in bacteria.
C. starch in animals.
D. glycogen in plants.
E. glycogen and starch in both animals and
plants.
A
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Which of the following statements is not true of nitrogen-fixing bacteria?
A. They live in root nodules. B. They are very common in symbioses with plants from the legume family. C. They are very common in symbioses in all plant families. D. Some are free-living in the soil. E. They provide an important way that biologically available nitrogen enters ecosystems.
A community refers to ____
a. the interaction between populations and their environment b. the interaction between species and their environment c. an area in which a single species lives d. all species that live and interact in a specific area e. the nonliving components of an area
Your friend wants to know why patients that receive organ transplants have to take immune-suppressing drugs for the rest of their lives. You tell her that:
1.The transplanted organ will only work properly with the immune-suppressing drugs. 2.The MHC markers of the donor organ do not match the patient, so the patient’s immune system will reject the organ without the medication. 3.The donor organ has a different MHC marker, which will attack and kill the patient’s cells if the medication isn’t taken regularly. 4.The medication is needed so the patient will not get sick after the transplant, which could cause the new organ to not function properly.
Medicinal chemists that synthesize drugs to kill pathogenic protozoans aim to target a novel mechanism to promote selective toxicity. The reason there are fewer antiprotozoal drugs compared to antibacterials is that ________.
A. there are few novel molecular mechanisms in protozoa compared to human hosts since both are eukaryotes, so toxicity is an issue B. protozoans are not pathogenic compared to bacteria so developing drugs is not cost-effective C. most antiprotozoal drugs also kill bacteria so they are not given their own category D. targeting novel mechanisms in protozoans is far more costly than it is for bacterial cells