Fragile X syndrome is a human disease caused by a trinucleotide repeat expansion that results in loss-of-function of the FMR1 gene on the X chromosome. An animal model with most features of this syndrome could be created by
A) adding a transgene containing a mutant copy of the FMR1 gene to a mouse or primate genome.
B) adding a transgene containing a wild-type copy of the FMR1 gene to a mouse or primate genome.
C) knocking out both copies of the FMR1 gene from a mouse or primate genome.
D) knocking out one copy of the FMR1 gene from a mouse or primate genome.
C) knocking out both copies of the FMR1 gene from a mouse or primate genome.
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