A client undergoing conscious sedation is at risk for ineffective breathing patterns. Which of the following nursing interventions will help the client demonstrate a normal breathing pattern?
A) Reinforce use of incentive spirometer.
B) Encourage progressive activity.
C) Encourage the client to drink plenty of fluids.
D) Encourage the client to take deep breaths and cough at least every hour.
D
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A 40-year-old patient had an annual physical exam with routine lab tests. The patient's lab values include the following:
Hgb 13.8 g/dL Hct 40% BG fasting 146 mg/dL Triglycerides 200 mg/dL Total cholesterol 231 mg/dL LDL 175 mg/dL HDL 30 mg/dL The client was counseled to begin an exercise and diet regime for his health. After implementing healthy lifestyle changes 6 months ago, follow-up lab tests were drawn. Which of the following lab values indicate that the changes have been effective? A) Triglycerides elevated to 210 mg/dL B) LDL level decreased to 120 mg/dL C) Hgb decreased to 12.3 g/dL D) HDL increased to 35 mg/dL
A patient has been taking cholestyramine (Questran) to treat hyperlipidemia type II. The patient reports abdominal cramping and constipation
The patient's serum low- density lipoprotein (LDL) has decreased from 170 mg/dL to 110 mg/dL, and triglycerides have not changed from 150 mg/dL since beginning the medication. The provider changes the medication to colesevelam HCl (Welchol).The patient asks the nurse why the medication was changed, and the nurse will explain that colesevelam HCl is ordered for which reason? a. It has fewer side effects. b. It has more convenient dosing. c. It provides greater LDL reduction. d. It provides greater triglyceride reduction.
When you care for a patient on oxygen therapy, you should
A. Keep the head of the bed flat. B. Use petroleum jelly to lubricate the lips. C. Provide frequent oral hygiene. D. Move the patient quickly through the patient's activities to save energy.
Which of the following effects is an acidic drug such as aspirin likely to produce in the body?
A. No change due to upregulation in the number of transporters for tubular secretion B. Metabolic alkalosis due to limited number of transporters for tubular secretion C. Metabolic acidosis due to limited number of transporters for tubular secretion D. No change due to downregulation of the number of transporters for tubular secretion