A client with herpes simplex virus (HSV) presents to the clinic stating, "I can stop taking my drugs because I'm cured. My lesions are gone."
The health care provider bases his or her response knowing the pathophysiology behind the disappearance of HSV symptoms infection relates to:
A) Replication of the squamous epithelium
B) Periods of latency in the nervous system
C) Inhibition of cell-mediated immunity
D) Production of exotoxins
Ans: B
Feedback:
The clinical course of HSV infection is a product of the virus's ability to remain latent in the dorsal root ganglia for long periods. Latency refers to the ability to maintain disease potential in the absence of clinical signs and symptoms. When latent, the immune system is ineffective, but the virus does not actively inhibit the cell-mediated immune system. HSV does not produce exotoxins, and replication of squamous epithelium accounts for the warts caused by HSV.
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