Why did you perform multiple differential tests on the same three colonies for the Normal and Heat treatments? If you were able to observe your samples under a microscope, what conclusions can you make about the types of microorganisms that make up the colonies you observed?
What will be an ideal response?
Answer: We generally perform multiple differential tests on same colonies to differentiate among them and to identify the correct microorganisms forming colonies by studying morphology of the microorganism along with the colony characteristics.Heat treatment gives us information about the spores.Also,in staining processes heat treatment prevent the washing off of the microorganisms.
Microscopic observations help us to distinguish between different types of bacteria like gram positive and gram negative.We can know about the size,shape and overall morphology of the microorganism using microscopy.Further the colonies that we see on media along with the morphological characteristics that we have observed under microscopy helps us to identify which microorganism is actually present in the give sample.
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In an Hfr strain, the F episome is integrated between genes A and B. When this Hfr mates with an F? strain, gene B is always transferred first to the F? cell. What is the last bacterial gene that could possibly (at least in theory) be transferred to the F? from the host chromosome?
A) a gene that is 90 minutes from gene A B) a gene that is at 30 minutes from gene A C) gene A D) a gene that is at 60 minutes from gene A
Fumarate production is an oxidation reaction similar to the reactions that produce ?-ketoglutarate, and oxaloacetate, but with one key difference. What is it?
a. Fumarate production releases a water molecule. b. Carbon dioxide is released in this reaction. c. This reaction reduces FAD instead of NAD+. d. Fumarate production occurs twice per cycle.
In humans, urea and uric acid are produced by the kidneys
____________________ Indicate whether the statement is true or false.
To become active, Cdk1 must
a. bind cyclin. b. bind cyclin and be phosphorylated on tyrosine 15 and threonines 14 and 161. c. bind cyclin and be phosphorylated on tyrosine 15, and be dephosphorylated on threonines 14 and 161. d. bind cyclin and be phosphorylated on threonine 161, and be dephosphorylated on threonine 14 and tyrosine 15.