What is the primary regulator of the magnitude of alveolar ventilation under normal circumstances?
A. the H+ concentration in the brain extracellular fluid, which is monitored by central chemoreceptors
B. The PO2 of the arterial blood, which is monitored by central chemoreceptors
C. The PO2 of the arterial blood, which is monitored by peripheral chemoreceptors
D. the H+ concentration in the arterial blood, which is monitored by central chemoreceptors
E. stretch receptors in the lung
A
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Indicate whether the statement is true or false
What prevents the dramatic increase in venous return that occurs during exercise from damaging the cardiac myocytes?
A. The exercise demand on the heart causes it to stiffen, preventing overstretch. B. The pericardium acts as a limit on the amount of stretch. C. The increased heart rate limits filling time. D. Venous return is not sufficient to stretch the heart very much.
_________ membranes are associated with the lungs.
A) Peritoneal B) Pericardial C) Pleural D) Synovial E) Cutaneous
Damage to the type II pneumocytes of the lungs would result in all of the following except
A) a loss of surfactant. B) a decreased rate of gas exchange. C) increased surface tension in the alveoli. D) increased tendency to alveolar collapse. E) decreased number of dust cells.